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Fig. 2 | BMC Cardiovascular Disorders

Fig. 2

From: HiPSC-derived cardiomyocyte to model Brugada syndrome: both asymptomatic and symptomatic mutation carriers reveal increased arrhythmogenicity

Fig. 2

Characterization of hiPSC-CMs. (A) Allelic discriminations between the WT and mutated SCN5A alleles in asymptomatic and symptomatic CMs. (B) Immunocytochemical staining’s where red represents troponin T, green sodium channel Nav1.5 and Nav2.1 and blue DAPI-staining for nuclei. Scale bars 100 μm. (C) Relative gene expression levels of cardiac related genes. The GAPDH was used as an endogenous control. No statistically significant differences in expression levels of WT, asymptomatic and symptomatic CMs.

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BMC Cardiovascular Disorders

ISSN: 1471-2261

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