Endothelial nitric oxide synthase Asp298Glu (894G/T) gene polymorphism as a possible risk factor for the coronary slow flow phenomenon among Iranians

Table 4 Multivariable logistic regression analyses of the possible predictors of CSFP in the study population

Variables	OR (95% CI)	P value
Presence of allele ‘T’ of the eNOS 864G/T polymorphism	0.46 (0.11–1.98)	0.29
Presence of allele ‘T’ of the IL-1β 315C/T polymorphism	0.47 (0.13–1.7)	0.24
Interaction of eNOS 864G/T and IL-1β 315C/T	1
Age	1.08 (1.03–1.12)	< 0.01
Gender	0.22 (0.08–0.62)	< 0.01
Smoking	1.17 (0.48–2.83)	0.73
FH of CAD	2.15 (0.92–5.03)	0.07
DLP	1.16 (0.51–2.63)	0.72
DM	0.49 (0.19–1.25)	0.13
HTN	0.74 (0.33–1.64)	0.45
FBS	1.0 (0.99–1.01)	0.15
Cr	2.51 (0.4–15.77)	0.32
LDL-cholesterol	1.01 (1–1.03)	0.05
Hb	1.10 (0.81–1.50)	0.52
WBC	1 (1–1)	0.96
Platelet (× 10³)	1.0 (0.99–1.01)	0.79
ESR	1.01 (0.96–1.05)	0.77

CSFP coronary slow flow phenomenon, FH of CAD family history of coronary artery disease, DLP dyslipidemia, DM diabetes mellitus, HTN hypertension, FBS fasting blood sugar, Cr creatinine, LDL low-density lipoprotein, Hb hemoglobin, WBC white blood cell, ESR erythrocyte sedimentation rate

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