Skip to main content

Table 1 Baseline patient characteristics

From: Regional contributions to left ventricular stroke volume determined by cardiac magnetic resonance imaging in cardiac resynchronization therapy

 

Patients (n = 65)

Controls (n = 20)

Women

19 (29%)

8 (40.0%)

Age (years)

67 ± 9

62 ± 11

BSA (m2)

2.0 ± 0.2

1.9 ± 0.2

Diabetes

9 (14%)

0

QRS-duration (ms)

167 ± 17†††

95 ± 11

LBBB

56 (86%)

N/A

Heart rate (beats/min)

67 ± 13

62 ± 7

NIBP (mmHg)

  

 Systolic

127 ± 17

131 ± 13

 Diastolic

76 ± 9

75 ± 7

Medication

 

0

 ACEI/ARB

65 (100%)

 

 Betablocker

58 (89%)

 

 Diuretics

56 (86%)

 

 Antihyperlipidemics

41 (63%)

 

 Platelet aggregation inhibitors

35 (54%)

 

NYHA Class

 

N/A

 NYHA Class II

20 (31%)

 

 NYHA Class III

41 (63%)

 

 NYHA Class IV

4 (6%)

 

Etiology

 Ischemic

32 (49%)

 

 Non-ischemic

33 (51%)

 

LVEDV (ml)

326 ± 115†††

163 ± 37

LVESV (ml)

246 ± 110†††

66 ± 21

LVSV (ml)

80 ± 23††

97 ± 20

LVEF (%)

26 ± 8†††

60 ± 5

LV mass (g)

170 ± 49†††

112 ± 32

CO (l/min)

5.3 ± 1.5

6.0 ± 1.2

LVAVPD (mm)

8.3 ± 3.2†††

15.3 ± 1.6

SA area (cm2)

51 ± 11†††

34 ± 7

SVlong% (%)

53 ± 18††

64 ± 8

SVlat% (%)

41 ± 16†††

29 ± 7

SVsept% (%)

0 ± 15†††

10 ± 4

LGE positive

40 (62%)

N/A

  1. Continuous variables are expressed as means ± SD and categorical values in absolute numbers and proportion in parenthesis
  2. ACEI, angiotensin-converting enzyme inhibitor; ARB, angiotensin II receptor blocker; BSA, body surface area; CO, cardiac output; DCM, dilated cardiomyopathy; IHD, ischemic heart disease; LBBB, left bundle branch block; LV, left ventricular; AVPD, atrioventricular plane displacement; EDV, left ventricular end-diastolic volume; EF, left ventricular ejection fraction; ESV, left ventricular end-systolic volume; SV, left ventricular stroke volume; NCC, non-compaction cardiomyopathy; NIBP, non-invasive systemic blood pressure; NYHA, New York Heart Association; SVlat%, lateral contribution to stroke volume; SVlong%, longitudinal contribution to stroke volume; SVsept%, septal contribution to stroke volume
  3. P < 0.05 patients vs healthy controls
  4. ††P < 0.01 patients vs healthy controls
  5. †††P < 0.001 patients vs healthy controls