Skip to main content
Fig. 2 | BMC Cardiovascular Disorders

Fig. 2

From: Trimetazidine attenuates pressure overload-induced early cardiac energy dysfunction via regulation of neuropeptide Y system in a rat model of abdominal aortic constriction

Fig. 2

Comparisons of myocardial expression of fetal genes related to myocardial hypertrophy induced by AAC: results of real-time PCR analyses. a, Effects of AAC and TMZ pretreatment on mRNA levels of MHC-β in myocardium of rats from each group; b, effects of AAC and TMZ pretreatment on mRNA levels of ANF in myocardium of rats from each group. AAC abdominal aortic constriction, TMZ trimetazidine, ANF atrial natriuretic factor, MHC-β β myosin heavy chain; n = 4~6 per group, and samples were analyzed in triplicate. *, p < 0.05 compared with sham group at the same time point; #, p < 0.05 compared with AAC group at the same time point

Back to article page